Monday, 2nd December 2019.
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Welcome to lactate.co.nz
A business of BM&S Imports, New
Operating since 1988; A registered NZ
company since 1994
Kerry James Goodhew – Owner / Manager
Since 1995 on the launch of the world’s first
small hand held lactate test meter we have been
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Wednesday, 13th November 2019
I’ve loved the work of Peter Janssen for 30 years.
In particular his text “Training Lactate
Pulse-rate” which the equally world leading Polar HR Monitor company
took on and re-printed upto at least a 6th edition in 1995, the
current copy I have!
I can still pick it up and the
penny will drop on something, or, it is just so long since I first read it
that I am not quite re-membering and it is as per
the first time!
This tidbit highlights the amazing adaptability of an
individual (any of us!) from untrained to trained with respect to HR and
Scroll down or click here!
Wednesday, 13th November 2019
aware: Office closures!
Office will be closed for
processing orders on …
Tomorrow, Thursday 14th November, from
Midday. Re-open Monday 18th
Thursday 5th December, from
Midday. Re-open Monday 9th
Friday 20th December, closed. Re-open Monday 6th January
Thursday 12th March 2020, closed. Re-open Monday 16th March
Go to old notices: Archive: click!
Scroll below for today’s
“lactate testing tidbit”
Lactate testing tidbits…
December 2, 2019
Individual example: The primary purpose of
training, to improve one’s power “bandwidth”, is demonstrated by the much
greater range from one’s lowered resting heart rate to a much higher HR lactate
threshold deflection point.
This human adaptability
(change in performance potential) is dramatically demonstrated in this
(graph) of a 20 year old’s improvement before and
after a training period.
The max. HR does not change, no “improvement” to
be found there!
You can see that when Threshold was at 130, after
training this point of work, speed, or duration limitation will be akin to
“jogging”, being a new very extensive tolerance to duration of activity at that
pace-HR. Previously 130 HR would have
been entirely carbohydrate (glycogen) burning, strictly performance duration limiting
when untrained, lucky to last an hour let alone 90 minutes, the latter being a
typical athletic exhaustion of carbo substrate at Threshold.
After training, this individual at 130 HR is now
likely primarily fat-burning, moving at same or faster speed, but can go on theoretically
Tuesday, November 12, 2019
“Marius” on getting it
right: What lactate testing is all about
“Earlier studies done by Frank Evertsen and the renowned physiologist Saltin showed that the dominating Kenyans did much of
their training right below their Lactate Threshold (LT – right when you start
to accumulate lactic acid) as compared with Scandinavians who trained either
too easy or too hard with little “feel” for how hard they were running.”
Bakken, Norway, March 2000.
is plenty of considerable value on Marius’ wordpress
Games 2001 // 13.09 5000 –
800 m 1.51.19 (Oslo 97)
1500 m 3.38.86 (Rieti 05)
2000 m 5.01.46 (Florø 03)
3000 m 7.40.47 (Brussels 01)
5000 m 13.06.39 (Rome 04)
10000 m 28.26.x (Lisbon 00)
10000 road 28.26 (Oslo 00)
§ Olympic Games 2000/2004
§ World Championships 99/01/05
§ European Championships 02/06
§ European U23 99, European Juniors 97
§ 9th place World Championships 01
§ 12th place World Championships 05
§ Silver medal European U23 99
Marius has a “100 day Marathon Plan” designed for “anybody”
It has 15 instructional
videos, 10 additional videos, and 120 content packed pages!
It is …
“Structured so it is easy to customize for any runner
– regardless age, gender, speed, or skill”
Thursday, 16th May 2019
…(Shorty) is all concentration
on his way to winning his local Taranaki triathlon several years ago.
For a dose of motivation
whatever your chosen sport go here…
Friday, 8th December 2017
Again, apology for my sport bias but it
is the one I am most familiar with from my youth and the principles are
applicable to many sports!
When I came out of Phys Ed School in ‘83 I
went back to rowing a couple years later to experiment on myself with what
I’d learnt. On arriving, University
“Easter Tourney” rowing was not what it is today, feeling it mickey-mouse,
so I didn’t bother with it for 4 years, proving costly when trying to get
“back on the oar” with any kind of representative ambitions. Was a so-called adult student going in,
coming back to career hunting and rowing at 26.
I became increasingly fascinated with the
concept of Power-to-Weight as a determinant of performance. Soundly frequently defeated by about half
a dozen of the countries top Light-weights
(<72.5 kg), being only a “medium-weight” myself with a best racing (and
health) weight of about 82kg, I had to also contend with about a dozen
ahead of me in the Heavy-weight Single Scull category. Did okay with placings in Intermediate and
Noticed improved condition through a Season
coincided with becoming very trim with reduced weight (from around an
off-season 88kg, back in the day when there was such as off-season!). Was performance improvement just a factor of
improved cardio-vascular function or, and, if both, was it about carrying
less weight? … which had the most impact?
Then there was the phenomenon of Philippa
Baker and Brenda Lawson “pocket rockets” who for years annihilated any
truly heavy weight women at the NZ Champs and won individual world titles
in Light Weight and Under 23 respectively, and then took on the world
together reigning for several years in the Double Sculls.
It was their performance that inspired me
to create a power-to-weight performance table that gave predictive on-water
single scull times … (requested data for only on “even” courses, meaning no
tail-wind or current) … which was taken from one’s bodyweight and 2000m
Ergometer score. I put a survey to
NZ’s top rowers who kindly allowed use of their data (which could not
personally identify them anyway) and using mine and other modest performers
the spread on the table from average to superior was completed. It was
developed on old Concept2 B Models. I suspect the algorithm has changed
somewhat from those days so that for such a performance predictive table to
be used today it would need to be re-done for today’s “E” model and
superior technology in faster carbon-fibre skiffs.
We were only just transitioning from wooden oars in my-day!
The impressive German immigrant Herman Krutzman residing in Cambridge, himself an accomplished
sport scientist, Olympic level coach, and builder of skiffs branded in his
own name, Krutzman, was an incredible source of
additional “real” education for this “newbie” in hydrodynamics revealing
the massive increase in resistance
(additional force required from what is a modest “biological engine”) for
the increased surface area of a skiff due to increased weight.
If a variety of people today sent me their
best Single race time (again, even conditions only), with their Erg 2K PB,
and their bodyweight for their on-water performance I could modernize this predictive table. I have used it for good motivational
effect when I was coaching about 20 years ago!
want the old table as it
is, I will see if I can dig it out, drop me an email, or if you have a collection
of old NZ Rowing mags it appeared there around the late ‘80’s (think it
Then there was the spectator confusing
sensational Cohen and Sullivan, multi-World and 2012 Olympic Double Scull
Champions, medium-weights and average height (just like me!) that resumed
an interest in this Power-2-Weight thesis. When they stood on the dais against those they had defeated they appeared “little guys” at
around 6 foot! They also paced their
races in a style which was uncommon (subject for another “lactate tidbit”
article one day!). They would appear to sit modestly in the field to finish
over the last 500m with a blistering sprint that put fans on the edge of
seats, exhilarating, as they carved through the field.
This “Power-2-Weight” subject had its
original roots way back to a particular graph in
the now classic undergrad “Textbook of Work Physiology” by the legends Astrand and Rodahl (2nd Ed, 1977) that lends
to this (above) “story” to this day.
Traditional graphs of VO2max show that the
heavier you are the greater likelihood that you have the highest VO2max.
However, if you put a spread of people from those with light bodyweight and
relatively low VO2max against heavier people with higher VO2max you do not
necessarily have a predictor of performance in times, or across a finish
line, in cycling, running, or on water events!
However! The lower graph shows you that you do have
a predictor of performance if you correct for bodyweight by a function of
This means that instead of the all too common
standard expressions of simple litres per minute
VO2max or milliliters per minute per kilogram, use milliliters per minute per kilogram to -2/3.
it evens out (or accounts) for the effect of bodyweight on VO2max leaving
you an assessment of just who has the better VO2max irrespective
Why this third formulation is not used as
one standard in High Performance sport I have no idea, maybe someone can tell
me. (Maybe I’ve been away from ivory towers too long! ;-) It has only been around as a
clearer predictor than straight VO2max or milliliters per kilogram since
the leading-light sport scientists of the ‘60’s! Sure, Lactate Threshold is a better “fitness”
indicator, but VO2max assessment still has the place for predicting the
ultimate performance capacity and international competitive potential
(rank) of an individual.
So what practically is the value of all
this long commentary for you? How do you use this to improve performance?
the subject for Part Two!
Will try and get onto this before or over
the Xmas break. If you don’t hear from
me before, or I you, YOU HAVE A GREAT XMAS!
KJ & ER Goodhew
BM&S Imports – lactate.co.nz
Thursday, 28th September 2017
Please excuse this writer’s bias …an old rower!
NZ Rowing has hard-worked
its way to one of, if not “the” most spectacular result winning status of
any sport in Aotearoa. It amazed me the flagellation after the Olympics. To
me what I saw was all this young talent coming on with the Men’s 8 “up
there” just 7 seconds off Gold ….and the Women, “my god” I said …the women
coming 4th, just 4 seconds off the winner USA.
If each one of that crew has just
improved their condition, on average through the crew, to delay lactate
accumulation at wattage (power) or speed giving a 4 seconds improvement,
then we have not just the possibility of a medal but gold.
Even though I spent most of my years with
preference in small boats, the prospect of little ol’
NZ crashing the party in the Big Boats again after all these years has this
old-oar excited beyond what any (non-rowers) could believe.
Kia kaha NZ
women's eight crew in action in Florida Photo credit: Getty
24th January 2017
testing and illness or over-training syndrome.
In 1995 for the first time having on-hand
a small portable lactate testing device there was an explosion of thoughts
and enquiries on what else lactate testing could tell us apart from basic
determination of “performance at lactate threshold”.
I have forgotten who wrote that mood
turns dark a day to several days before an athlete physiologically plummets
into serious overtraining syndrome and performance capacity reversal, which
as we all know, the arresting of such performance collapse often cannot be
halted and can just carry on getting worse!.
I still wonder how many coaches and
trainers are mercilessly and uselessly thrashing athletes into poor
One approach to mitigating this was exemplified
by Rushall and Pyke in “Training for Sports and Fitness”, 1990, have
athletes fill out a “Daily Analysis of Life Demands”, which provide a
“window” into the state of the athlete.
From 1995 I tested myself (relatively
young compared to now!) and several others under the duress of severe
training demands walking the tight-rope of gain or
collapse as everybody felt they must do:
A simple submax
protocol was used: 20 minute easy warmup followed by 5 or 10 minutes at a
relatively easy below La threshold pace and lactate sample. If all was well the HR and La at set pace
would stay the same over a week or so, and then as weeks went by reveal
state of the athlete in either of 3 ways:
1. HR and La stay the same at set pace, or …
2. … they drop, hinting an improvement in
functional capacity, or …
3. … if things are either about to go bad or
have gone bad for the athlete the HR and La are higher than usual for set
The lactate sampling submax
test could be done weekly and does not stress and strain an athlete, it can
become just part of a training session!
A simpler HR version of this (without La test)
can be done everyday! …to confirm the kind of
work the athlete is up for that day.
You do not have to thrash athletes with
all-out tests to reveal that their capacities are on the improve, or in
jeopardy, or that they have stalled, hinting at it being time to review the
work programme due to stagnant adaptation, or the
athlete just drifting without purpose !
An unexpectedly raised HR and La at submax can indicate another matter of concern that
competes with training adaptation and that is energy for the immune system:
Our lactate meters are used in
veterinary clinics and A&E dept’s for sepsis screening: Infection raises your lactate level: A resting reading at or above what would be
your Work Threshold would be of concern!
However also, I have found some sport
supplements will (strangely!) raise your lactate level above what you would
expect from having been resting! I
have not looked for research into what you eat as negatively affecting
lactate, work capacity at La.4. If
there is still nothing on this since I looked in 1995 there is an
opportunity for a student!
Just some observations! Hope you like and will look
into it further for yourself, to build on this!
4th April 2016.
correlative observation on the success of NZ Olympic sports that use (or do
not use) lactate testing.
Since first introducing convenient small
portable (hand-held) lactate testing to NZ sport in 1995 I’ve noticed that
the sport codes that involve a significant endurance component, who have
used our lactate testing product the most over the past 21 years, have been
the most successful.
Those that buy the most lactate testing
strips are hands-down unquestionably our most successful sports. Other endurance sports who one might think
would use lactate testing to be more successful are not successful.
Just a simple observation!
18th December 2015.
Maintaining Nose Breathing
and Lactate Threshold
In 1994 I came across this book by Douillard, intrigued not the least reason since it
mirrored my business’s name.
In it was his thesis on the
great benefits of nose-breathing over the all too common mouth-breathing
during exercise, or worst of all open-mouth breathing as normal while
It was not something I had thought about
but subsequently realised that I was a somewhat
good nose-breather during moderate exercise.
From this point on I insisted upon it
with myself and after the period of adaptation to it, receiving clear
benefits, advised everybody who came through my door to convert to it also.
It varies between 3 weeks to 3 months for
people to adapt to strict adherence. Douillard’s thesis
is that your physiology is changed. I thought that lactate testing should show up some of this adaptive process.
I experimented with it in self-training
for Masters Rowing and did pretty good with results, avoiding the frequent over-training
of younger years, generating the not too uncommon thoughts in one’s
accumulated years of “what if I knew
then what I know now?” C’est la vie!
From long memory looking back,
self-experimentation with this gave equally intriguing lactate results.
1. In the beginning of trying this, the upper-end
of maintaining nose-breathing was a very modest pace, along with expected
Heart Rate. (Many clients had to cut
back to almost a fast-walk and could barely handle staying with it. A lot
of them arguing against it!) Lactate
at “nose-threshold” in myself then was only about 2.5 to 3 mmol. Above this intensity I would burst
into old-habit mouth-breathing.
2. As the weeks went by HR at pace dropped and
lactate went to about 3.5 mmol at
max. nose-breathing with an increased pace at this. The HR drop at pace was not due to any
increase or other factors of altered training over that year or any other
years. That is, I had not really had
a drop in HR at set paces for similar amount and level of conditioning in
3. After many years from 1994 “living” this
way, the last surprise result was that I could maintain nose-breathing
(with some will - effort) at a pace-HR that equated to 4 mmol Threshold.
That was some years ago now and I remember thinking that I felt I could
probably taken it up to
maintain nose-breathing at just above Threshold, say 5 mmol. Have not to this day verified if that is
there is an experimental idea for a post-grad thesis!
There is a lot more to this but can’t put
a book here!
All the best to you for Xmas
KJ & ER Goodhew.
4th November 2015.
A relatively small amount of what is
initially high intensity tolerance work repeated daily with a largely
untrained individual has a dramatic adaptive effect on improved tolerance
to that work as measured by lactate response.
Referring to the left
hand graph, there is as said for the “tidbit” below (last week), so
much in this. Edwards (1939) is
another giant in early Exercise Physiology that the profession is built on
“Fast forward”: Being the original
importer-distributor of small hand-held lactate testing meters in 1995, the
“Accusport” by Boehringer-Manheim, we wanted to
gather as much broad information as possible on the possible varieties of
use and benefits of such easy technology. In those days we offered the “BM&S Clinic” for personal
training in any sport code, individual or team, and a specialised
weight-loss service. Memory from
those days is that successes with people in weight-loss were at least if
not more gratifying than successes with athletes!
One client’s success that stands above
all was a 136kg woman who had “tried everything” to lose weight and
despaired. Included with
“everything” were gyms and personal trainers: Their approach was to “thrash” and
basically hurt her with relentless high intensity sessions that were
unpleasant. An objective
“professional” approach in weight-loss exercise prescription simply was not
there in those days. (Being away from involvement in such services for
about 12 years I have not really looked closely at the state of the
efficacy of the “weight-loss game” today!)
What did we do then that was different?
We lactate tested everybody who came through our door whether their goal
was prospective Olympian, beginner runner wanting to do first marathon,
weight-loss, triple by-pass cardiac rehab, wanting to look good for her
wedding, a horse, …and even the postman! True!
When my favourite
client story first came through the door she was scared
of exercise. I told her that she would lose weight with walking at
correct intensity and that it would be easy compared to what she had done
and that we would work it out by testing her blood to make sure the
exercise is not at unpleasant and unnecessary high intensity which puts
damaging acid into your blood and through your body. That is, putting
things into simple terms without off-putting sport science jargon.
As a prelim., with HR monitor on we set
off up the road which leads to Frasers Gully about 2.4 km in length with a
more challenging slope in its last ¼, but which is in the most part a
barely perceivable gradual slope to find that she could not walk around
100m without bursting into rapid mouth breathing and appearing
bothered. Next time I conducted a
necessarily “off-the-cuff” modified lactate assessment protocol just to
find where her 4 to 5 mmol HR intensity was.
Several times per week we met to do
Frasers Gully interval style whereby as soon as her HR climbed above her
original 5mmol HR we would stop, let it drop for a minute or two and then
on until the alarm called for the next stop.
At the end of just one month my 136kg
client who had lost about 6kg walked to the top of the gully without one
stop or alarm of high HR. Her blood
pressure, borderline before, was now normal.
She told me she was “…amazed how easy and
pleasant the exercise intensity was…” for her to achieve so much.
The graph on the left in today’s copy
& paste reminded me of the mechanism of this favourite
23rd October 2015. : FYI: If you thought I was talking crazier than usual,
some kind of load-up error occurred where the wrong table appeared! Corrected
Classic tabulated data on responses to various
brief training impulse/recovery cycles from the now classic eponymous text
by Astrand and Rodahl, “Textbook of Work
Yes! I still have my copy from undergrad
days! In it is this referenced
original study by Christensen et al, 1960!
I’ve added to the right an approx.
conversion of mg/100ml to mMol.
There is so much that can be taken from
this table that I cannot really get into too much here since having looked
at it and making use of it in training individuals years ago. In past days “in da game” I got a reputation
for taking what people perceived as quite ordinary ranked athletes to a
level unexpected. One day watching
an athlete come in a spectator said to me, “…how is that possible”? I was
young then, saying somewhat flippantly, “training secrets”.
This was later not taken well being accused of offering unfair advantage to
the athlete over others in the club who were expected to dominate in a
predicted procession of race positions. Envy amongst coaches in NZ sport is
a not uncommon terrible thing; I did not last long in that club!
How did I use this table? The issue for the average athlete is that
they have average aerobic capacity, average VO2max, and come up against
gifted athletes with lungs and heart of a horse. What is one to do? We know VO2 is trainable to a significant
degree, but more so is % VO2 that AT kicks off a rapid lactate accumulation
as determinant of endurance performance.
All we can do is optimize the average athlete’s VO2 and then shift
their “threshold” as close as possible toward their VO2max. (…plus effective
race strategy, work on motivation, psychology, and structural soundness –
anatomy – for freedom from potential to injury. The latter most important
since however you do it, VO2max pace work is high stress work. I don’t
advocate you do any of this with a beginner!)
We also know that anaerobic-lactate work
destroys aerobic enzymes for upto several days and opens one up to illness
(immune compromise). You can see
from the table that doing VO2max pace continuously for 4 minutes takes
lactate to 16.7 mMol and therefore aerobic
enzymes destroyed. This is not suitable training! Coaches who over max test or over-race
their athletes in this form are simply stuffing them up!
You can see for an already fairly well trained athlete work/rest intervals of 10:5 and
15:10 elicit too high lactate response, destroying aerobic capacity for
You can see the best strategy to elicit a
near maximum VO2 of 5.3 with a lactate response equivalent to “going for a
jog” is 15:15.
Experiment with this and during it wack in
a lactate test or two to determine your athlete’s individual response to
I can assure you this works a treat. I self-tested (when I was young
with “OCD” for discerning “secrets”) and used it on numerous athletes to
raise them up quickly. If you
schedule it weekly use their mood to decide whether to stick with it
religiously as per schedule or not.
Go to Archive of Lactate testing “tidbits”: Click!
Link will be live soon!
Do you have a story for us, maybe research you are
doing? We love to hear and share your stories.
Comparing the old with the new!
or Team Trainer/Coach use
and health prescription (Green prescription)
- Sepsis screening
faster, takes just 15 sec’s
smaller sample required
& Software available, connectivity to PC.
“The original” (1998)
- Oldie but a goodie!
Ø Child-birth – foetal
hypoxia screening – Maternity Wards NZ DHB’s
- Sepsis screening
Sport Science/Medicine; Personal or Team Trainer/Coach; Olympic sport teams
and health prescription (Green prescription)
Currently LP (1) test strips remain
available until further product development!
Tuesday, 12th November 2019
1. Shipment is in!
Backorders went out same
day, last Friday, 8th, so you should have yours by now.
If your order is on “PBD discounted” we’re awaiting your
payment before sending!
are within a year to Olympics
Please help us to help you by not running out of your own
store of strips before re-ordering. You know most of the time we can meet
your urgent requirement, but it does happen that we could run out for a
month or so, any time, in the run up to Olympics!
Pricing this shipment fractionally lower for Carton Quantity
purchases on “PBD discounted” terms.
new lactate testing tidbit:
Check-out below sage advice on LT-training
by middle-distance track athlete Norwegian Marius Bakken
Nb: His page not attended for a long time since he
has gone “doctoring”, his final post appearing to be 2010, but with his
comments on Kenyan and Moroccan dominance still ultimately relevant today!
Thursday, 16th May 2019
Update: Tuesday, 28th May 2019
All backorders out
Wednesday and Thursday 22nd / 23rd, so you should have
had yours by the next day! Let me know if otherwise; Unless we’re awaiting
on your payment for orders at payment
before dispatch additional discount pricing.
now: Stock is going fast!
A month of your backorders has cleaned out half the shipment.
Trend continuing, we could be out of stock again in a month or two: That’s
4 to 6 months’ supply going double quick! This could mean near future 2 months out of stock as we cannot
adjust forecast at factory quicker than 4 months out!
3. Keep your own minimum stock:
you don’t need them now, best you order now rather
than later to keep a minimum level of your own back-up stock on-hand over
the next year. Surging demand before Tokyo Olympics appears to be upon us
See Shorty Clark’s “story” below
how he has used our two spearhead products LactatePro
and TANITA BIA monitors to great
advantage to go “10-in-a-row at
Kinloch NZ Championships” in an outstanding Masters
career. Click here: Shorty!
Monday, 4th March 2019
CLOSED: Thursday and Friday, 7th and 8th March inclusive.
place orders by end of trade Wednesday, before 4pm, or if “PBD” discounted
price terms are wanted, place order by midday so that we have time to see
your payment come through.
back on-board Monday 11th March.
Trajectory of demand shows that we will run out of stock before the next
shipment gets here in April.
Do not wait to order only when you need them, hold your own
minimum supply. The long upto 18 months to expiry facilitates
for you to do this. Especially important to keep your own minimum supply if
you have important scheduled testing programmes. Please manage your own
minimum stock level right through to the starting guns firing, Olympic-time
next year, Tokyo-2020
Extended again: Monday,
4th March 2019
at landed cost pricing” on LP-Meters is back for Term 1!
previously exclusive “educational offer” is now extended to anybody wanting
3 or more LactatePro2 Meters. Only genuine enquiries will be engaged with
for this pricing, therefore we’ll be somewhat
for classic teaching of experimental method utilising a sport science
context, suitable from senior High School educational levels onward.
Meter and strips can occupy 3 students being the Experimenter- recorder of
data and timing, The Tester takes the blood samples calling out numbers to
record, and The Subject who undertakes a work protocol on say a Treadmill,
Exercycle, or Rowing Ergometer. A
before and after Treatment can be engaged in, for example baseline data
followed by the effect of 8 weeks Training.
of 15 can be occupied with 5 Meters.
Alternatively 10 of your sport science
(physical education) students can be occupied with 5 Meters if your
students are sent off to recruit a Subject from outside of their class,
e.g. someone from the school rowing, athletic, or cycling squad.
this to your 2018’s year budget TODAY!
Monday, 23rd January 2017
trend since Rio carries on!
ordered 50% more Meters than we ever have with 2/3 gone either before they
got here or within 2 weeks.
Amazing! At current trend we
are going to run out before next shipment gets in, don’t procrastinate!
confidential deal on sets for educational exercise physiology (or other)
labs is available: Minimum quantity
= 5 Meters.
Such a minimum
quantity was chosen since it could meet the small number of students in
labs of size 10 to 15 students with one each of tester, recorder, and
Secondary “Sport Science” students could do this with tester, recorder, and
roping in a subject from say the school rowing squad!
a personal view: All those in their
last year of school rowing wanting to maintain their interest and progress
in the sport would have their decision making greatly helped with a clear
lactate profile revealing performance improvement progress and potential
trajectory which is much better shown with a lactate profile rather than
all-out Erg tests which can wildly disguise capacities and potential for
us for the Educational Quantity Deal
on 5 (or more!):
Enquire now: Email (click here!)
Monday, 4th April 2016
We are still looking for a few more “On-sellers” (retailers) in
various areas around NZ: Contact us:
NB: Below is “C&P”
from original “My TANITA” posting here … www.bodyfatmonitors.co.nz/#Shorty
The focus on the My TANITA
testimonials is on body composition results through the season but for Shorty
there are almost as many references to lactate test results!
Original posting: Tuesday,
18th February 2014
Latest update: Wednesday, 27 March 2019
…(Shorty) is all concentration
on his way to winning his local Taranaki triathlon several years ago.
Update: Wednesday, 27 March 2019
As I becried
my own paucity of self-updates on my own profile (text well above), the
purpose of content meant to give you an idea how to use your TANITA for a
healthier (“wealthier”) life, I am so pleased to get back into action on this
page to give you also an update on how long-time friend and stand-out athlete
Shorty Clark has progressed since
first post in February 2014. Scroll to
the bottom of this panel to view that original post!
Shorty is a “real athlete”
by comparison to myself who “plays around” with a few goals and achievements
per year: Anyone wanting to achieve in their sport, at whatever age you
happen to be, could not do better than to emulate Shorty’s clear focused
approach that I’ve observed for at least around 20 years now. When I launched
small portable lactate test meters
on the NZ sport market in 1995, sometime soon after I believe Shorty was the
first individual athlete of any sport-code to get one and self-test. He has
done lactate threshold testing ever since and still a couple times per year
puts through his values for lactate at paces to me. Since it was so long ago
(you know what memory is like!) the only triathlete who may have beaten him
to it happened to be a Sports Medicine Doctor and a Triathlon-NZ coach!
Shorty was also the first
genuine athlete in NZ to embrace TANITA
body composition analysis to carefully monitor his condition (physique) to be
in peak readiness for championship competition. Being the importer, in position of
nation-wide observer of users, it stood out that Shorty was 15 years ahead of
most sport codes getting with it on the objectivity of what TANITA can offer a
top athlete, and it still ever amazes myself that after all this time (near ¼
century!) some New Zealand sport codes still have not got with the programme of clear benefits and advantages to using
this world-leading technology! I have known many athletes in my now long
sport related and side-line coaching and personal training career, but I have
known few to match the intellectual approach and dedication of Shorty Clark.
For all my own observational experience I’ve not seen anything like it, his
career has been simply amazing to watch. I find him inspiring and we can all
do with a bit of that sometimes to get us up and going. 😉
Shorty often keeps me
updated with Facebook messenger, so I have thought for simplicity in this
long overdue update I would just copy & paste some of his quick reports
to me (without my responses as most did not add anything!): Hope you find
these as interesting as I did?
These below are snapshots of
Shorty’s path from success at National Champs – Olympic Distance, Mt
Maunganui to a record at NZ short-course championships, Kinloch.
New Plymouth Tri-Athlete a
Master at using TANITA
In February 2019 Shorty Clark went 10 in a row at Kinloch
Did 63km Bike followed by a 13km Run. Was 2hrs 30mins on bike, ave 25.0kph , @ 115, and Run was
05.12pkm ave ,@ 139 . LACTATE was 1.6 mols after
03.00. Unreal!!!. Feeling pretty dam good.
the National Champs Olympic Distance was HUUUUUMUNGUS at the Mount. Great to
beat my mate, finally. Best race for Mental attitude and tenacity. Felt so
good. Lactate 4:6mols after 01.30.
way to get ahead of my competitors. If you don't change, how can u win!!!. Now for the next 9
weeks of stepping back up to the plate and getting it right for NZ Sprint
Champs, Kinloch, 10 Feb.
National Sprint Champs, Kinloch Sunday 10th Feb. Going for the big one!!!! =
10 in a row is the goal, undefeated !!!!. No
pressure -haha. Massive 2hrs Compu Ride yesterday
at 95% , 05.00am !! then at 5.00pm cranked a 8km Run
out . Merry Xmaz I said to myself -haha
Yes , sea swim 2km only in my NEW WETSUIT !!!.
question brand of wetsuit!) Yip the ELITE of the Elite.=
Huub, Agilis, Brownlee . Named after the Brownlee
start to 2019.
Great sensible eating at Xmaz time is paying early
good 13km hilly run today with max type efforts thrown in when going up
hills. WAS 68.00, Ave 05.15pkm, @143, cad 87, .
LACTATE 2.4 mols!!.
Base work is a must , and i
feel that its the total glue to a top performance. Auzzie Worlds proved that for me, as did winning the
National Olympic Tri Champs in December.
Build that " engine", as Adrenalin and Hype will turn on hidden
POWER and SPEED.
You're a Champion, and that doesn't go away, so
"mount up and get back on the horse". Success does await you for
and Run are STRONG!!!, and so rapt about the Run. Cranked a Mini tri myself
on saturday= 1.750-45km- 6km, = 02:40.00. Was at 85%
swim 95% Bike 100% Run. Lactate = 6.7mols. Excellent quick recovery. Bike and
Run, hilly course, and ave Run 04:31pkm @ 157, Cad=
92. Stride = 1.20mts.
nicely to Kinloch National Sprint Champs.
good after a Rest Day today. Cheers.
Rippa of a tri session yesterday
1.600, 28km and 6km. 2hr neat. LACTATE 5.0 mols. Swim 100%, Bike 98% and Run
100%. Hilly course. Ave 04.35pkm Run. Top quick RECOVERY. Feel bloody good
and reckon that " cream" is right there.
off plan:) Easy sea swim. Massage session 3.00pm. Tues nil Wednesday nil.
Thurs small swim pool on drills Then leave thurs
arvo for Hamilton. Swim pm with Coach squad, easy stuff. Fri 6.30am squad
swim. 10.00 leave for Kinloch. Sunday = the bull stops haha
the guns off 10.04am.
got the nz no. 1 spot at kinloch.
won it by default, as rules says you must be a nzer
(for NZ Champion title). (But) beaten by my pommy mate, but he cannot be
awarded it, so i naturally go from 2nd to 1st.
my worst ever race too. went in with bad elbow injury- super inflammed arm and elbow. was a survival type mode race
for me. a super dumb accident on the tues before.
still inflammed and swollen. got x rays monday, awaiting results
to say LACTATE was 8.3!!! at race FINISH.
Shorty Clark Male 65-69
Geoff Martin Male 65-69
Warren Taylor Male 65-69
NB: I will do a review
later of the meaning of various measures (trends) in Shorty’s body
composition results, to maybe help you with what it means!
You, like myself, do not have to be a standout Champion like Shorty Clark to
benefit greatly from TANITA, but if you decide to, you can go all the way to
your very own personal bests, measured and proven by the world’s best
health-fitness technology! - KJG
Wednesday, 19th February 2014
concentration on his way to winning Taranaki Triathlon several years ago…
…uses LP2 to good effect.
Approximately 12 months ago (2012)
I started using my Lactate Pro 2 Meter for Training and Racing purposes. Used
another brand before that and previous to this I
relied heavily upon Heart-rate Monitors and “subjective feel” data to
ascertain my performances and success/failures.
The most crucial benefit I find is
that it allows me to train and race way more precisely and accurately. It
gives me the added advantage of knowing how to increase my Lactate Tolerances
when in heavy/extended sessions or even when doing a recovery type mode
work-out. I can therefore correlate my Heart-rate a
lot closer to my Lactate Thresholds, thus allowing me to race smarter,
quicker, efficiently and faster.
When racing, it’s critical not to
start at to higher Heart-rate and Lactate level, as
this generates into fatigue, poor performance and the inevitable bonking. By
measuring and testing your Lactate levels in training, you are then able to
maximise your performance and results, come race day. It’s such a valuable
and necessary tool in my training and racing programme, that without it, I
would be always wondering and 2nd guessing at why my performances were not at
their highest level. One thing that is for sure – Blood Lactate levels DO NOT
lie!!, even though you may think and feel differently.
The Lactate Pro 2 gives you that
complete and 100% answer in how to improve your training speeds, conditions,
results and performances, via the easy and simple testing methods, plus the
added benefits of being simple to test, quick on results, with accuracy and
lightweight to use/carry.
I thoroughly recommend the Lactate
Pro2 Meter as the best, overall piece of training equipment that has lifted
my performances, and allowed me to compete at my top
potential. As a serious Age Group Triathlete, the Lactate Pro 2 has given me
an added advantage to optimise my full athletic potential,
and permits me to train / race in the best possible shape and
Mens 60 - 64 Age Group, Tri New
Arkray Website News Release
The smallest meter in its class just
got easier to use. Even better performance for ARKRAY’s card-sized blood
lactate measurement device
Inc. (Headquarters: Kyoto City, Nakagyo-ward), a manufacturer of sample
test devices and in vitro diagnostic reagents is set to launch its new
and simple blood lactate meter, the Lactate Pro™ 2
LT-1730 on 17 April. In its card size form, it is small enough to
allow measurement anywhere and boasts increased performance for the rapid
measurement of lactate.
in blood is an essential marker in the fields of sports physiology, sports
science and training. The simple and rapid testing of lactate is a big
benefit to both research and medicine.
answer the needs of the market, ARKRAY released the Lactate Pro TM back in
February 1997, which marked a significant reduction in size for blood lactate
at the time. This new card sized device enabled simple measurement of lactate
anywhere and helped to support clinical testing across many different
is now set to release the simple blood lactate meter, the Lactate Pro™ 2 LT-1730
together with its compatible reagent/ measurement electrode, the Lactate Pro™ 2 Sensor. The
measurement time has been cut by 75%* to just 15 seconds; the minimum sample
volume reduced by over 90%* to just 0.3µL and the basic performance of the
device improved significantly. The hassle associated with correction of
reagent lots has been eliminated which also helps to reduce the risk from
will continue to answer diverse testing needs in the market.
comparison to existing ARKRAY products:
Lactate Pro™ 2
Pro™ 2 Sensor
Main features of the Lactate Pro™ 2
●A simple blood lactate device, the smallest of its kind
Palm-sized for measurement in any environment: now you can measure lactate
●Minimum sample size: just 0.3µL
Development of a new reagent sensor: measurement possible with just 1/10th
the volume previously needed
●Measurement time: 15 secs
On site measurement is possible using finger-stick blood: results are shown
in just 15 seconds from application of blood to the sensor- one quarter the
time needed for measurement with previous devices.
●No correction needed
No more hassle from correction of sensor lots (insertion of sensor chips
before measurement): this also helps to reduce the risk from correction
Blood lactate is often used in the scientific training for endurance sports
such as soccer, swimming and athletics. As the intensity of the exercise
increases, so too does the concentration of lactate in the blood. By
measuring this change, training can be configured to delay sharp rises in
blood lactate. (There are also Medical
and Equine/Veterinary applications – KJG, BM&S Imports-NZ.)
Lactate Pro™ 2 LT-1730
2. Release date
17 April 2012 (Tues.)
Lactate in blood
LOD enzyme electrode
15 sec/ sample
Min. sample vol.
Lactate Pro™ 2 Sensor (Electrode
for measuring lactate in blood)
Ext. output function
internal temperature sensor
Temp: 5-40 C, Humidity
20-80% RH (No condensation)
3V lithium battery/
50mm(W) X 12mm(D) X
Approx. 45g (Incl.
Do you need an
“ordinary” Bathroom scale or a Kitchen scale?
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Ø GP’s and
specialist Sports Medicine Dr’s
Ø All the
/ Obstetric Wards
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Ø All the
major University, Polytechnic, and College of Education Sport Science and
Nutrition service provider & research dept’s.
Ø NZ Academy
relevant NZ Olympic and other sport organisations (directly, or indirectly
Coaches, Trainers, Sport Scientists.
self-testing athletes, youth age through world masters age competitors
research dept’s (e.g. Lactate testing of salmon!)
Health & Fitness
FIND A TESTING SERVICE
This article posted here 26 Nov. 08 cited from HIV InSite, University
of California, San Francisco.
lactate testing predicts mortality of severe sepsis in a predominantly HIV
type 1-infected patient population in Uganda
UCSF Institute for Global Health Literature Digest
Published April 17, 2008
Moore CC, Jacob ST, Pinkerton R, Meya DB, Mayanja-Kizza, Reynolds SJ, et al.
lactate testing predicts mortality of severe sepsis in a predominantly HIV
type 1-infected patient population in Uganda. Clin Infect Dis 2008 Jan 15;
To evaluate the ability of a handheld portable whole-blood lactate (PWBL) analyzer
to predict mortality in patients who are admitted to the hospital with severe
A prospective observational study.
An accident and emergency department of Mulago Hospital, a national referral
hospital in Kampala, Uganda.
72 patients were enrolled in the study. Inclusion criteria were ≥18
years of age and admission to a medical ward, along with: 1. two or more
systemic inflammatory response syndrome criteria (body temperature, >38°C
or <36°C; heart rate, >90 beats/min; respiratory rate, >20
breaths/min; or peripheral WBC concentration, >12,000 cells/mm3 or
<4000 cells/mm3; or thermodysregulation; 2.
systolic blood pressure ≤100 mm Hg; and 3. a suspected infection. Exclusion
criteria included acute cerebrovascular events, gastrointestinal hemorrhage, or admission to the surgical or obstetrics
and gynecology ward.
There was no intervention in this study. This analysis was conducted among a
subset of 253 patients recruited to study the incidence, management, and
outcomes of sepsis. From this sample, the first and last 50 consecutively
enrolled patients were recruited to determine the predictive value of PWBL in
predicting mortality from sepsis. Background information - including age,
sex, HIV-1 serostatus, and prescribed antiretroviral medicines (ARVs)-was
recorded. At patient enrollment, temperature, heart
rate, respiratory rate, and blood pressure were measured. To determine
outpatient survival, an attempt was made to telephone patients 30 days after
their discharge from the hospital. A rapid HIV-1 test and malaria smear were
performed at Mulago Hospital. A local private clinical laboratory provided
results of lactate and bicarbonate analysis. PWBL was obtained using a lancet
to collect a drop of whole blood from the patient's finger, for analysis by a
handheld portable device. This instrument uses enzymatic determination and
reflectance photometry of lactate in the plasma portion of whole blood using
a measurement strip. Standard laboratory serum lactate (SLSL) concentration
was obtained by phlebotomy for venous blood samples. Within 2 hours of sample
collection, the sample was transported in a standard serum tube via a cooler
to the clinical laboratory, where blood was centrifuged and serum was removed
for use in the lactate assay.
Information was available for 72 of the 100 enrolled subjects. The mean age
of participants was 35.7, 61.1% were women, and 81.9% were HIV infected, with
a mean CD4 lymphocyte count of 88.6 cells/ mm3. These 72 patients were similar to the larger study population in age (mean age,
35.7 vs. 33.8 years), sex (61.1% vs. 59.1% female), HIV-1 seropositivity
(81.9% vs. 86.6%), and ARV status (13.9% vs. 10.6% ARVs prescribed).
Fifty-nine (81.9%) of 72 evaluated patients were infected with HIV-1. The
in-hospital mortality rate was 25.7% (18 of 70), and the in- and outpatient
mortality at 30 days was 41.6% (30 of 72). PWBL was positively associated with
in-hospital but not outpatient mortality (p <.001). The receiver operating
characteristic (ROC) area under the curve for PWBL was 0.81 (p <.001). The
optimal PWBL concentration for predicting in-hospital mortality (sensitivity,
88.3%; specificity, 71.2%) was ≥4.0 mmol/L. Patients with a PWBL
concentration ≥4.0 mmol/L died while in the hospital substantially more
often (50.0%) than did those with a PWBL concentration <4.0 mmol/L (7.5%)
(odds ratio, 12.3; 95% confidence interval, 3.5-48.9; p <0.001). SLSL
levels were lower among survivors than among deceased. The ROC under the
curve for predicting in-hospital mortality was 0.72 (p=0.004). SLSL results
were inconsistent and less predictive of mortality than were those of PWBL.
The authors conclude that PWBL testing can quickly identify patients who
require immediate interventions, and it should be included in
evaluation and treatment algorithms for septic patients. PWBL testing could
be used in village health posts, for earlier transfer of septic patients to
facilities with a higher level of care, and in referral hospitals, for triage
of patients to acute care settings where appropriate resuscitation can begin.
Elevated lactate concentrations and poor clearance of lactic acid are known
to increase mortality from severe sepsis. Lactate concentrations, combined
with other laboratory measures, are used to guide early management of sepsis.
Measurement of lactate concentration in developing countries is difficult
because of limited resources. An effective, inexpensive method to measure
lactate concentration without sophisticated laboratory resources has the
potential to offer a method to identify patients in greatest need of rapid,
aggressive treatment of sepsis.
The use of the PWBL and its value in predicting mortality from sepsis in
populations with high prevalence of HIV has potential to assist health care
workers in resource limited settings in prioritizing these patients.
Hospitalization, availability of support for potential multisystem failure,
and rapid administration of antimicrobial agents can be delivered more
urgently in persons with high lactate concentrations.
This article posted here 26 Nov. 08
cited from Lab Tests On-line.
Testing in Acute Assessement
What is being
This test measures the amount of lactate in the blood or, more rarely, in the
fluid. Lactate is the ionic (electrically charged) form of lactic acid.
It is produced by muscle cells, red blood cells, brain, and other tissues
energy production and is usually present in low levels in the blood. Aerobic
energy production is the body’s preferred process, but it requires an
adequate supply of oxygen. Aerobic energy production occurs in the
mitochondria, tiny power stations inside each cell of the body that use
glucose and oxygen to produce ATP (adenosine triphosphate), the body’s
primary source of energy.
When cellular oxygen levels are decreased,
however, and/or the mitochondria are not functioning properly, the body must
turn to less efficient anaerobic energy production to metabolize glucose and
produce ATP. In this process, the primary byproduct
is lactic acid, which can build up faster than the liver can break it down.
When lactic acid levels increase significantly in the blood, the affected
person is said to have first hyperlactatemia and then lactic acidosis (LA).
The body can often compensate for the effects of hyperlactatemia, but LA can
be severe enough to disrupt a person’s acid/base
(pH) balance and cause symptoms such as muscular weakness, rapid
breathing, nausea, vomiting, sweating, and even coma.
Lactic acidosis is separated into two types: A
and B. Type A may be due to inadequate oxygen uptake in the lungs and/or to
decreased blood flow (hypoperfusion) resulting in decreased transport of
oxygen to the tissues. The most common reason for this is shock from a
variety of causes including trauma and blood loss, but LA may also be due to
conditions such as heart
heart failure, and pulmonary edema (fluid in
the lungs). Type B is caused by conditions that increase the amount of
lactate/lactic acid in the blood but are not related to a decreased
availability of oxygen. This includes liver
storage diseases (such as glucose-6-phosphatase deficiency), drugs and
toxins, severe infections (both systemic sepsis
and a variety of inherited metabolic and mitochondrial diseases (forms of
muscular dystrophy that affect normal ATP production). Strenuous exercise can
also result in increased blood levels of lactate.
Testing in General Practise
With the advent
of the world’s first small hand-held lactate test meter launched in 1994 by
Boehringer Mannheim, the use of lactate prescription for effective and
objective “Green Prescription” was already well considered. My personal observation is that the Europeans
are well ahead of us in this medically correct and safety orientated approach
to intensity prescription of exercise. It is this correct medical approach that
has been begging for implementation here in NZ. Fourteen years later (2008)
the right Doctor with knowledge & motivation to champion this cause here is yet to be forthcoming!
To view larger image
in viewer on your PC click here.
2. This article posted here 26 Nov. 08 cited on-line at Wiley Interscience.
Interval versus continuous training in patients with severe
COPD: a randomized clinical trial Eur Respir J 1999;
R. Coppoolse*, A.M.W.j. Schols*, E.m. Baarends*, R. Mostert**, M.a. Akkermans**, P.p.
Janssen**, E.F.m. Wouters*
**Astmacentre Hornerheide, and *Dept of Pulmonology, Maastricht University, the
Correspondence: A.M.W.J. Schols
Dept of Pulmonology
P.O. Box 5800
6202 AZ Maastricht
Fax: 31 433875051
information is available regarding the physiological responses to different
types of exercise training in patients with severe chronic obstructive
pulmonary disease (COPD). The aim of this study was two fold: firstly, to investigate the physiological
response to training at 60% of achieved peak load in patients with severe
COPD; and secondly to study the effects of interval (I) versus
continuous (C) training in these patients.
patients with COPD (mean±sd
forced expiratory volume in one second: 37±15% of predicted, normoxaemic at rest) were evaluated at baseline and after
8 weeks' training. Patients were randomly allocated to either I or C
training. The training was performed on a cycle ergometer, 5 days a week, 30
min daily. The total work load was the same for both
resulted in a significant increase in oxygen consumption (V 'O2)
(17%, p<0.05) and a decrease in minute ventilation (V 'E)/V 'O2
(p<0.01) and V 'E/carbon dioxide production (V 'CO2)
(p<0.05) at peak exercise capacity, while no changes in these measures
were observed after interval training. During submaximal exercise a
significant decrease was observed in lactic acid production,
being most pronounced in the C-trained group (-31%, p<0.01 versus
-20%, p<0.05). Only in the I-trained group did a significant increase in
peak work load (17%, p<0.05) and a decrease in
leg pain (p<0.05) occur. Training did not result in a significant
improvement in lung function, but maximal inspiratory mouth pressure
increased in both groups by 10% (C: p<0.05) and 23% (I: p<0.01).
The present study
shows a different physiological response pattern to interval or continuous
training in chronic obstruction pulmonary disease, which might be a reflection of specific training effects in either
oxidative or glycolytic muscle metabolic pathways. Further work is required
to determine the role of the different exercise programmes and the particular category of patients for whom this might be
Equine Trainer & Veterinary
Davie is probably the pre-eminent advocate in Australasia of effective training
of race horses incorporating lactate testing. The great value of his book is the
simplicity of its explanation. Anybody
can use it to catapult the training of their horses into 21st
Century method to earn positive performance improvements.
Price NZ$ relative to currency x-rate!
Bad news for heavy riders and narrow
March 3, 2008
Researchers in the US have bad news for overweight horse riders. A
study has found that horses that have to carry
between 25 and 30 per cent of their bodyweight have more physical problems
related to exercise than those who carry 20 percent or less.
Horses carrying 30% body weight showed a significant increase in
muscle soreness and muscle tightness scores. The changes were less marked
when they carried 25% body weight.
Dr Debra Powell .... conducted a study ....performing
a standardised ridden exercise test in an indoor school arena. After five
minutes active walk to warm up, the horses were ridden at a trot (3m/s) for
4.8km, followed by 1.6km at a canter (5m/s). This exercise schedule was
chosen to simulate a 45-minute work period of work typical of an
intermediate-level riding school horse.
The researchers measured heart rate, plasma lactate
concentration and creatine kinase. Lactate is produced in the muscles during
exercise. At low levels of work the body can metabolise it and so levels in
the plasma remain low. As the work level increases the rate of lactate
production exceeds the body's ability to remove it and so concentrations
rise. Creatine kinase (CK), an enzyme present in the muscles, is released
into the blood as a result of some types of muscle damage.
... Plasma lactate levels were higher immediately after
exercise and 10 minutes after end of exercise, in horses that carried 30% of
their body weight....
Health, Fitness & Sport
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Product Services: Lactate
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